You need to lose weight – which means you have to drastically reduce calories so you will always be hungry and lose lean mass.
The MaV method, eliminates these two problems utilizing the principles of ketogenic died combined with Nutrigenetic testing to calibrate the maintenance phase of the nutritional plan specifically for you nutritional profile, based on over 90 years of combined scientific research.
Hello, my name is Marie Vranceanu, I am a biologist and nutritionist specializing in the ketogenic diet and Nutrigenetics.
I used to be obese.
I struggled years to reach a normal weight, trying every diet imaginable.
Nothing worked. In my despair I decided to study Nutrition and completed my Masters in Italy at Universita degli Studi, Milan, and later at the University of Florence for my specialisation in nutrigenomics.
You can see my linked profile here: Marie Vranceanu – Linkedin
Now I help people lose weight, even if they have failed with other diets and lost any hope of success.
The MaVDIET or MaVMethod is the result of 15 years of school and practice, more nutritional congresses and courses than I can count, and my own personal experience with my body and more than 1000 patients that I have personally treated.
This is my work.
I believe that Nutrition is the foundation of health.
Being overweight is a threat to health. Eating incorrectly for your nutritional profile can lead to a multitude of other chronic health problems.
And “Nutrition” is more than “eat a salad”
Nutrition is very complex – and as I have learned nutrition is meaningless if it is not personalized – just like medicine.
You can not treat every patient the same. It just doesn’t work.
The MaVDIET has two phases when applied for weight loss:
First the weight loss phase:
- 8 weeks of Lipoketofast
- Detox Week
- 1 -2 weeks of intensive ketogenic fasting treatment with supplemental microfiltered whey protein combined with a specialized formula of amino acids to stimulate natural human growth hormone production and prevent lean muscle loss while targeting adipose tissue in hormone dependent areas. For women this is the hips, buttock and belly. In men it is the belly.
Particular attention should be paid to the choice protein supplement to be used because the numerous products offered for sale do not always correspond to a protocol so delicate as MaV diet.
The product used must adequately stimulate GH secretion, be pure and not have excess carbohydrates. Quantities must also be calculated for each patient.
- 6 week of progressive reintegration based on your Nutrigenetic profile
At the end of this stage, my patience generally loose 15 kg (33 lbs) or more depending on how much they have to loose.
The maintenance phase based on the GenoMaV Nutrigenetic test
Obviously the spectacular results obtained during the first phase need to be maintained, not only for aesthetics – but also to prevent disease.
Most maintenance programs fail because they are not personalized in a scientific way.
Nutrigenetic testing solves this problem.
A DNA sample is taken by oral swab after which the sample is sent for analysis at our genetic lab in Italy. The results are available in about 30 days.
The genetic test that work was conceived by prof. Dr. Keith Grimaldi, one of the most prominent nutrigenetisicts in the world. You can see his linked in profile here.
If you are interested in the MaV method – either for weight loss or for developing a personalized nutritional plan for your genetic profile for your own life long health please contact me here and we can explore some personalized options based on your goals. Nutrigenetic testing and consultation packages start at 375 US plus shipping.
In the struggle with extra weight, I tried diets over diets, some famous, but all proved unsuccessful. The method we have managed to weaken Marie Vrânceanu 15 Kg in eight weeks. If you want to loosen nice, I strongly recommend her method.
~ Dr. Laura Oşean
More about GenoMav Nutrigenetic testing
Over 30,000 genes make up our DNA, a kind of guide with instructions for our body
One thing is certain: our hereditary dowry can not be changed, however we can change our lifestyle.
Since the code was deciphered, we can interfere with the predisposition to certain pathological conditions by preventing their expression.
Problem: We all know that diets work for some of the most popular and are completely ineffective for others.
Some individuals suffer from hypertension, although following dietary salt restriction, while others have high cholesterol even consuming a minimum of fat.
Solution: Understanding individual genetic variability
Nutrigenetics is able to provide personalized nutrition, calibrated based on your genetic profile.
It is a diet not only helps permanent loss of weight, but also can prevent pathologies to which an individual is genetically prone.
A calorie is the same for everyone, but there are plenty of other aspects to be taken into consideration. We know that many genetic variations can influence how we absorb and metabolize food and these variations play a role in determining dietary regime that works for each individual person.
Nutritional genetics, between myth and reality ….
Not all nutrigenetic tests are created equal.
There are those who sell tests claiming that they would be able to provide information on the diet model that should be followed diet (Atkins, Zone).
There is no scientific evidence to that effect.
There is no “absolutely healthy” food, but food more or less suited each of one us.
What we do know, based on extensive scientific evidence it is that there are genes that may influence how we absorb and metabolize carbohydrates, fats and how our body responds to exercise.
For example, products resulting from digestion will be transported from the gut into the blood. There is a protein that transport fats in the blood and because of genetic variations, effectiveness may vary from person to person.
If efficiency will be higher, the person will get more calories from the same amount of fat compared with another carrier to work more slowly. This topic alone could be the subject of many dissertations.
We also test for genetic polymorphisms that exert an important role in inflammatory processes of detoxification, antioxidant activity, insulin sensitivity, heart health and bone and definitively verify predisposition to celiac disease (gluten intolerance) , lactose and caffeine
Nutrigenetics is the latest generation of science that focuses on the individual genetic characteristics by relating food with the bodies metabolism, the living environment and individual predispositions. More specifically, it seeks to identify those small yet vital individual SNP genetic variations that can lead to undesirable responses from the body with the consumption of certain types of food.
If you are interested in the MaV method – either for weight loss or for developing a personalized nutritional plan for your genetic profile for your own life long health please contact me here and we can explore some personalized options based on your goals.
Nutrigenetic testing and consultation packages start at 375 USD.
1. LCT gene
Lactose is digested by the enzyme lactase. In many regions of the world presence decreases significantly with age, so digesting lactose becomes difficult. In Europe, lactase persistence of genetic variation determines which makes it possible to digest lactose in all stages of life.
Polymorphism C / C means that the person does not possess the genetic variant that allows durability of lactase, therefore will have insufficient capacity to digest lactose.
Diet = reducing or avoiding lactose.
2. ACE gene
ACE gene plays a key role in cardiovascular homeostasis. The gene polymorphism is present insertion type / deletion (alleles I-insertion, deletion allele-D) and influencing enzymatic activity.
Recent studies have shown the link between genotype I / I, I / D and sensitivity to salt.
Other tests showed that genotype I / I is predisposed to hypertension when salt intake is excessive. It is recommended to not more than 5.5 grams of salt per day, which corresponds to weight 2.2 grams of Na, the molecule responsible for the taste of salt, and its effect on the blood pressure.
Diet = reducing salt intake.
3. Gena ADH1C
Alcohol metabolizes alcohol dehydrogenase 1C forming acetaldehyde, the toxic compound responsible for the negative effects of excessive consumption. In turn, acetaldehyde is metabolized by the enzyme aldehyde dehydrogenase non-toxic compounds. GenoMaV testes for versions that produces changes in amino acid sequence that modify enzyme efficiency. For example, the homozygous genotype A / A alleles Ile (Ile-Ile), is characterized by the presence of isoleucine in a specific position of the sequence amino acid enzyme and has a catalytic efficiency metabolizing elevated alcohol much faster than Val genotype.
Diet = reduce the amount of alcohol.
4. APOC3 gene and gene LPL
Recent studies have shown an interaction between these two genes and nutrition. Apolipoprotein C3 is a very low density lipoprotein (VLDL) and contains mainly triglycerides. Lipoprotein lipase inhibits APOC3 (LPL) and slows catabolism of triglyceride. Genotype G / G APOC3 is associated with elevated levels of triglycerides. The result LPL gene (CC) contributes to changing the lipid profile, is associated with low levels of HDL (good) cholesterol and high levels of triglycerides.
Diet = recommended reducing dietary saturated fat below 16 grams per day, eliminating trans fats, replacing them with a larger quantity of olive oil.
5. CYP1A2 * 1F gene = 5
Is coding for the enzyme CYP1A2 cytochrome P450, involved in phase I (activation) the elimination of toxins – such as cancenigenics of smoked meat and – and metabolism of caffeine. The genotype of the alleles homozigoză A (AA) coding for the enzyme activity rapidly and therefore would be activated quickly prepared meat potentially toxic substances at high temperatures.
Diet = drastic reduction in the consumption of grilled meats and smoked maximum once a week. As for caffeine, this form of the enzyme metabolizes well.
GSTM1 gene, coding for the enzyme glutathione S-transferase isoenzymes is a family detoxified toxic molecules that catalyze the conjugation with glutathione different in order to make them less reactive and more easily eliminated from the body. GSTM1 gene polymorphism type insertion / deletion, I / D, causes loss of function enzyme.
Diet = increased consumption of cruciferous vegetables.
7. IL6 gene and TNF gene
IL6 and genaTNF gene, coding for pro-inflammatory cytokines homonyms are involved in the regulation of the immune response. The polymorphisms present in these genes, IL-6-174G / C and TNF-308G / A, affects the amount of cytokine produced. Polymorphism in the gene IL-6, GC, is the substitution of a G (guanine) and C (cytosine), resulting in a tendency of increase in the synthesis of the cytokine same name.
Diet = increasing the amount of omega 3
MTHRF gene coding for the enzyme is involved in the metabolism and use of folic acid and vitamins B6 and B12. The enzyme plays a central role in DNA synthesis and methylation. The genotype of 677C / T coding is correlated with less efficient enzyme associated with increased levels of homocysteine in plasma.
Diet = increasing daily intake of folic acid and vitamins B6 and B12.
9. PPARG gene and ACE gene
PPARG gene is encoding for a transcription factor that influences the glucose levels and insulin levels. Due to the presence or absence of polymorphism allele Pro12Ala influences gene expression, and therefore, the amount of protein produced. It has been shown that individuals who have heterozygous for alleles Ala have beneficial effects on blood glucose and insulin levels. For those with homogenous genotype alleles and genotype I / I ACE gene it is recommended to reduce consumption of refined carbohydrates and sugars, eating low GI foods (whole grains), not to exceed glycemic load of 80 grams per day .
Refined carbohydrate diet = reduction and glycemic load (GL) <80 g / day.
10. SOD2 gene
SOD2 gene, coding for the enzyme manganese superoxide dismutase MnSOD locatized in the mitochondrial matrix, is the first line defense cells against free radicals. MnSOD oxygen catalyze dismutated superoxide and hydrogen peroxide, thereby removing free radicals. Free radicals even if in evolution assisted in the maintenance of cellular homeostasis, are toxic molecules capable of inducing oxidative damage to biological macromolecules indiscriminately, and are primarily responsible for certain pathological conditions and aging.
The genetic test for this enzyme demonstrates heterozygosity.
Profile C / T is associated with an intermediate enzyme activity.
Diet = increasing daily intake of folic acid and vitamins B6 and B12.
VDR gene =
VDR gene coding for the vitamin D receptor is that affect the production of various proteins, some of which are involved the use of calcium. The heterozygous genotype for the allele C (CT), has been shown to influence the absorption of calcium and the bone structure.
Diet = increasing amounts of vitamin D and calcium.
Genetic variability affects how each of us responds to physical activity. Some people have to train more, some less, some need to focus on aerobic activity etc.
If you are interested in the MaV method – either for weight loss or for developing a personalized nutritional plan for your genetic profile for your own life long health please contact me here and we can explore some personalized options based on your goals. Nutrigenetic testing and consultation packages start at 375 USD.